whole mouse genome 4 × 44k oligo microarrays (Agilent technologies)
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Agilent technologies
whole mouse genome 4 × 44k oligo microarrays
Whole Mouse Genome 4 × 44k Oligo Microarrays, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/whole+mouse+genome+oligo+4_44k+microarray/pmc03672050-149-25-24
Average 90 stars, based on 1 article reviews
Whole Mouse Genome 4 × 44k Oligo Microarrays, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/whole+mouse+genome+oligo+4_44k+microarray/pmc03672050-149-25-24
Average 90 stars, based on 1 article reviews
whole mouse genome 4 × 44k oligo microarrays - by Bioz Stars,
2026-09
90/100 stars
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Microarray:Article Title: Transcriptomics and proteomics analyses of the PACAP38 influenced ischemic brain in permanent middle cerebral artery occlusion model mice Article Snippet: The gels were stained (8 μL of 10 mg/mL ethidium bromide in 200 mL 1X TAE buffer) for approximately 7 minutes and the stained bands were visualized with the ChemiDoc XRS+ imaging system (Bio-Rad, 6000 Alfred Nobel Drive, Hercules, CA 94547, USA). .. A Article Title: Microarray Analysis of Gene Expression Changes in Neuroplastin 65-Knockout Mice: Implications for Abnormal Cognition and Emotional Disorders. Article Snippet: One microgram of total RNA was amplified and labeled with a One-Color Quick Amp Labeling Kit (5190-0442, Agilent Technologies, Santa Clara, CA). .. The fluorescencelabeled cRNA was hybridized onto the Article Title: B cell hyperactivation in an Ackr4-deficient mouse strain is not caused by lack of ACKR4 expression. Article Snippet: 1Institute of Immunology, Hannover Medical School, Carl-Neuberg-Straße, Hannover, Germany 2Cluster of Excellence RESIST (EXC 2155), HannoverMedical School, Carl-Neuberg-Straße, Hannover, Germany Correspondence Prof.Dr.. ReinholdFörster,HannoverMedical School, Carl-Neuberg-Straße1, 30625 Hannover,Germany.. E-mail: foerster.reinhold@mh-hannover.de Abstract The majority of genetically modified C57BL/6 mice contain congenic passenger DNA around the targeted gene locus as they were generated from 129-derived embryonic stem cells (ESCs) with subsequent backcrossing to the C57BL/6 genetic background.When studying the role of atypical chemokine receptor 4 (ACKR4) in the immune system, we realized that the two available Ackr4deficientmouse strains (Ackr4−/− andAckr4GFP/GFP) show profoundly different phenotypes: Compared to wild-type and Ackr4GFP/GFP mice, Ackr4−/− mice show a strong accumulation of plasma blasts in mesenteric lymph node and spleen as well as increased B cell proliferation after in vitro activation. Gene Expression:Article Title: Transcriptomics and proteomics analyses of the PACAP38 influenced ischemic brain in permanent middle cerebral artery occlusion model mice Article Snippet: The gels were stained (8 μL of 10 mg/mL ethidium bromide in 200 mL 1X TAE buffer) for approximately 7 minutes and the stained bands were visualized with the ChemiDoc XRS+ imaging system (Bio-Rad, 6000 Alfred Nobel Drive, Hercules, CA 94547, USA). .. A Article Title: Microarray Analysis of Gene Expression Changes in Neuroplastin 65-Knockout Mice: Implications for Abnormal Cognition and Emotional Disorders. Article Snippet: One microgram of total RNA was amplified and labeled with a One-Color Quick Amp Labeling Kit (5190-0442, Agilent Technologies, Santa Clara, CA). .. The fluorescencelabeled cRNA was hybridized onto the Hybridization:Article Title: Microarray Analysis of Gene Expression Changes in Neuroplastin 65-Knockout Mice: Implications for Abnormal Cognition and Emotional Disorders. Article Snippet: One microgram of total RNA was amplified and labeled with a One-Color Quick Amp Labeling Kit (5190-0442, Agilent Technologies, Santa Clara, CA). .. The fluorescencelabeled cRNA was hybridized onto the other:Article Title: The role of transcriptional factor D-site-binding protein in circadian CCL2 gene expression in anti-Thy1 nephritis Article Snippet: The Article Title: D-site binding protein regulates cell proliferation through mediating cell cycle progression in rat mesangial cells. Article Snippet: Over proliferation of glomerular mesangial cells (MCs) disturbs mesangial homeostasis and leads to renal damage in mesangioproliferative glomerulonephritis.. It is documented that transcriptional factors may be involved in the proliferation of MCs.. This study aims to identify the key transcriptional factor that prevents the MCs from over proliferation and to clarify its regulatory mechanism. Article Title: Identification of Gene Expression Changes from Colitis to CRC in the Mouse CAC Model Article Snippet: The mouse expression profiles were scanned using an Article Title: Prenatal Alcohol Exposure Impairs the Placenta–Cortex Transcriptomic Signature, Leading to Dysregulation of Angiogenic Pathways Article Snippet: One-color comparative hybridization was performed using Whole Mouse Genome Oligo 4_44K Microarray (G2519F-014868, Agilent Technologies, Les Ulis, France) to compare gene expression profiling. cRNAs were synthesized from 100 ng total Labeling:Article Title: GATA 2 hypomorphism induces chronic myelomonocytic leukemia in mice Article Snippet: .. For microarray analyses, RNA quality was verified with an Agilent‐2100 Bioanalyzer; RNA was labeled with Cy3 using a Low Input Quick Amp Labeling Kit and hybridized to a Expressing:Article Title: B cell hyperactivation in an Ackr4-deficient mouse strain is not caused by lack of ACKR4 expression. Article Snippet: 1Institute of Immunology, Hannover Medical School, Carl-Neuberg-Straße, Hannover, Germany 2Cluster of Excellence RESIST (EXC 2155), HannoverMedical School, Carl-Neuberg-Straße, Hannover, Germany Correspondence Prof.Dr.. ReinholdFörster,HannoverMedical School, Carl-Neuberg-Straße1, 30625 Hannover,Germany.. E-mail: foerster.reinhold@mh-hannover.de Abstract The majority of genetically modified C57BL/6 mice contain congenic passenger DNA around the targeted gene locus as they were generated from 129-derived embryonic stem cells (ESCs) with subsequent backcrossing to the C57BL/6 genetic background.When studying the role of atypical chemokine receptor 4 (ACKR4) in the immune system, we realized that the two available Ackr4deficientmouse strains (Ackr4−/− andAckr4GFP/GFP) show profoundly different phenotypes: Compared to wild-type and Ackr4GFP/GFP mice, Ackr4−/− mice show a strong accumulation of plasma blasts in mesenteric lymph node and spleen as well as increased B cell proliferation after in vitro activation. |
